半岛bd体育手机客户端 介绍
靶标：
TCIRG1 半岛bd体育手机客户端 别名： ATP6N1C； ATP6V0A3； Atp6i； OC-116kDa； OC116； OPTB1； Stv1； TIRC7； Vph1； a3； TCIRG1； T cell immune regulator 1, ATPase H+ transporting V0 subunit a3； T cell immune regulator 1, ATPase H+ transporting V0 subunit a3； V-type proton ATPase 116 kDa subunit a 3； ATPase, H+ transporting, 116kD； T-cell immune response cDNA 7； T-cell, immune regulator 1, ATPase, H+ transporting, lysosomal V0 protein a； T-cell, immune regulator 1, ATPase, H+ transporting, lysosomal V0 subunit A3； V-ATPase 116 kDa subunit a 3； V-ATPase 116 kDa subunit a3； V-ATPase 116-kDa； V-ATPase subunit a3； V-type proton ATPase 116 kDa subunit a； V-type proton ATPase 116 kDa subunit a3； osteoclastic proton pump 116 kDa subunit； specific 116-kDa vacuolar proton pump subunit； vacuolar proton translocating ATPase 116 kDa subunit A； T-细胞免疫调节因子1(TCIRG1)； 背景信息：
This gene encodes a subunit of a large protein complex known as a vacuolar H+-ATPase (V-ATPase). The protein complex acts as a pump to move protons across the membrane. This movement of protons helps regulate the pH of cells and their surrounding environment. V-ATPase dependent organelle acidification is necessary for such intracellular processes as protein sorting, zymogen activation, and receptor-mediated endocytosis. V-ATPase is comprised of a cytosolic V1 domain and a transmembrane V0 domain. Alternative splicing results in multiple transcript variants. Mutations in this gene are associated with infantile malignant osteopetrosis.
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