閾剁墝浼氬憳绗?span style="font-size:1.5em;color:#f21">2骞滁/div> 璁块棶閲廁 3486863 缃戝潃:antibody.cnreagent.com 鍦ㄧ嚎鐣欒█
浜у搧鎼滅储
鎶椾綋 >> 鍏朵粬鎶椾綋
閽艰緟鍥犲瓙鍚堟垚铔嬬櫧1鎶椾綋
閽艰緟鍥犲瓙鍚堟垚铔嬬櫧1鎶椾綋鍥剧墖
浜よ揣鏈烔 1鍛?/span>
绱㈠彇璧勬枡鍙婃姤浠饵/span>
浜よ揣鏈烔 1鍛?/td>
浜у搧鍒悕: 閽艰緟鍥犲瓙鍚堟垚铔嬬櫧1(MOCS1)鎶椾綋
Anti-MOCS1
浜у搧浠嬬粛
闈舵爣锛欬p style="text-indent: 2em;">MOCS1


浜у搧鍒悕锛欬p style="text-indent: 2em;">MIG11锛 MOCOD锛 MOCS1A锛 MOCS1B锛 MOCS1锛 molybdenum cofactor synthesis 1锛 molybdenum cofactor synthesis 1锛 molybdenum cofactor biosynthesis protein 1锛 MOCS1A enzyme锛 cell migration-inducing gene 11 protein锛 molybdenum cofactor biosynthesis protein A锛 molybdenum cofactor synthesis-step 1 protein A-B锛 閽艰緟鍥犲瓙鍚堟垚铔嬬櫧1锛 閽艰緟鍥犲瓙鍚堟垚铔嬬櫧1(MOCS1)锛


鑳屾櫙淇℃伅锛欬div style="text-indent: 2em;">Molybdenum cofactor biosynthesis is a conserved pathway leading to the biological activation of molybdenum. The protein encoded by this gene is involved in this pathway. This gene was originally thought to produce a bicistronic mRNA with the potential to produce two proteins (MOCS1A and MOCS1B) from adjacent open reading frames. However, only the first open reading frame (MOCS1A) has been found to encode a protein from the putative bicistronic mRNA, whereas additional splice variants, whose full-length natures have yet to be determined, are likely to produce a fusion between the two open reading frames. This gene is defective in patients with molybdenum cofactor deficiency, type A. A related pseudogene has been identified on chromosome 16.

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娴撳害锛欱atch dependent (Please refer to the vial label for the specific concentration.)
绋€閲婃瘮渚婜 Optimal dilutions/concentrations should be determined by the end user
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